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Lee 1:30 Station K 2126 - Perioperative Outcomes of Combined Gynecologic Oncology and Urogynecologic Surgeries K. Tyson 1:30 Station L 2303 - Comparison of Laparoscopy and Laparotomy in Primary Cytoreductive Surgery of Advanced Epithelial Ovarian Cancer J. Keum Ii 1:30 Station M 1870 - Comparison of Laparoscopic Versus Open Radical Hysterectomy in Early Cervical Cancer after Completing Learning Curve and Reducing Intraperitoneal Tumor Exposure J. Kim 1:30 Station N 1538 - Incidence of Gynecologic Cancers in Women after Uterine Fibroid Embolization E. Paasche-Orlow 1:30 Station O 1251 - Opportunistic Salpingectomy at Time of Non-Gynecologic Laparoscopic Procedures Would Significantly educe varian Cancer Mortality and Would Reduce Overall Healthcare Expenditures. Mccracken 1:30 Station R 2615 - Robotic Tumor Debulking Off External Iliac Vessels for Management of Recurrent Ovarian Cancer L. Menderes 1:30 Station S 1354 - the Safety and cacy of ntra-Arterial Versus Intra-Venous Neoadjuvant Chemotherapy in Patients with Locally Advanced Cervical Cancer: A Meta-Analysis C. Zhang 1:30 Station T 2534 - Opioid Use and Misuse Among Gynecologic Oncology Patients S. Naumann 1:40 Station A 1868 - Prognostic Value of Preoperative Lymphocyte-Monocyte Ratio in Patients with Ovarian Clear Cell Carcinoma B. Lee 1:40 Station B 1471 - Single-Site Laparoscopic Total Hysterectomy and Bilateral Pelvic Lymphadenectomy for Endometrial Cancer Y. Johnston 1:40 Station D 2 - Survival utcomes of eoadjuvant Chemotherapy ollowed by aparoscopic or pen adical ysterectomy ersus Concurrent Chemoradiation in atients with ocally Advanced Cervical Cancer H. Ouh Laparoscopy 1:40 Station E 1206 - the Effect of Sub-Cutaneous and IntraPeritoneal Anesthesia on Post Laparoscopic Pain: A Randomized Controlled Trial O. Sagiv Endometriosis 1:40 Station F 1582 - Laparoscopic Excision of Endometriosis Does Not Reduce the Risk of Reoperation Within 2 Years Compared with Ablation of Implants M. Hua 1:40 Station H 2920 - A Retrospective Look at Gynecological Surgical Complications A. Jimenez Cabrera Urogynecology 1:40 Station J 1463 - Postvoid Residual Measurements by Bladder Ultrasound in Obese Women: Are They Accurate Alesi Surgical Technologies Applied Medical Avanos (Acute Pain) Baxter International, Inc. The result is a premier network of member ambassadors invested in learning and advancing minimally invasive surgery across the world. With an extensive inventory of parts, technical knowledge, and an in-house repair center, we offer our customers the quickest turnaround in the business. It is our mission to achieve this while also reducing healthcare costs and offering unrestricted choice. Applied is committed to advancing minimally invasive surgery by offering clinical solutions and sophisticated training, including workshops, symposia and our simulation-based training programs. Headquartered in Alpharetta, Georgia, Avanos is committed to creating the next generation of innovative healthcare solutions which will address our most important healthcare needs, such as reducing the use of opioids while helping patients move from surgery to recovery. The portfolio of products includes a comprehensive range of reusable and reposable gynecologic instruments such as needle holders, graspers, scissors and forceps. When you need Exhibitor Descriptions us most, our clinically differentiated surgical care products support hemostasis, tissue sealing, reconstruction, tissue repair, and intraoperative patient care. Our robust portfolio has been demonstrated to reduce intra- and post-operative complications that require costly blood transfusions and extend operating time. Less complications often translates into faster recovery for your patients and greater cost e ciencies for your hospital or clinic. Medical develops, manufactures, and markets differentiated surgical implants for the treatment of Stress Urinary Incontinence (Desara Sling System) and Pelvic Organ Prolapse (Vertessa Lite). Since 1, Mediflex has been innovating devices for surgical e ciency and retraction save time and cost, reduce staff and produce better surgical outcomes. Bolder Surgical was founded to revolutionize minimally invasive surgery by providing right-sized instruments that improve access and visibility.

Purulent nephritis of mixed type, from calf: a, microscopic abscess: b, embolus: c, normal urinary tubules d, urinary tubules devoid of their epithelium, containing coagulated material. When a purulent infiltration extends over a large area of subcutaneous, submucous or other loose cellular tis- 282 sue, Inflammation. Where the tissue soft- ening or the gravitation of the pus into dependent positions allows the exudate to escape the abscess if is said to break or perforate; and in such instances there are formed narrow canals in lined with pus and reaching stance to the surface these are called fistula. A loss of sub- upon the external surface or mucous membranes caused is by suppurative destruction of the tissue process as ulceration. The suppurating focus is, is encapsulated by this demarcating grozvth, that its it is enclosed in an abscess wall, which on inner surface usually has a grimy, grayish-yellow or slate-gray color. The involuntary muscle and elastic tissue of arteries exhibit marked resistance to purulent softening; in suppurative cavities, as of the lungs or udder or of a muscular tissue, vessels are often found as bridge-like Hemorrhagic, Gangrenous Inflammations. The vascular connective tissue which develops upon exposed suppurating surfaces (ulcers, surfaces of wounds) presents a reddish-gray color and a granular or finely nodular, uneven appearance {granulation tissue) purulent exudate arises from this also so long by cicatricial proliferation; as the microorganismal cause of inflammation continues active. This tissue serves to restore the tissue loss, filling out the cavity into scar tissue (cf. With multiplication of the pyogenic organisms in the affected tissue not only does the local suppurative inflammation become cor- respondingly prolonged, but there arises the probability of its exten- lymph spaces give rise to fresh inflammatory reaction in a constantly expanding area. The fact that the leucocytes have wandered into the suppurating area, may take up some of the bacteria and carry them some distance away, until stopped by the occurrence of paralysis and death and their transporting function thus ended, gives an excellent opportunity for the entrance of pus-producing germs into the lymph channels, lymph nodes and even the blood. The bacteria advancing along the multiplying organisms, over which the bactericidal forces of the blood have no influence and the phagocytic cells no power, into are thus likely to set up tatic new foci of suppuration, so-called metas- suppuration, in the lymph glands and any other places which they have been carried as emboli. We speak of hemorrhagic inflammation, where the exudate contains a notable admixture of red blood cells, and is consequently of a reddish, grayish-red to dark red, chocolate or cafe-au-lait appearance. Serous as well as fibrinous and purulent exudates may assume this character, which may be regarded as indicative of some especially severe disturbance of the vessel walls, which the blood current in in turn has occasioned the vessels, and marked slowing of the inflamed part, diapedesis of the erythrocytes or actual rupture of is therefore accompanied by stasis and haemorrhage. Putrefaction of the exudate and of the inflamed tissue (ichorous, gangrenous or putrid inflammation) is necessarily the result of the invasion of putrefying bacteria into the necrotic tissue and the masses of blood and exudate, dead material like the tissue itself. All chronic inflammations, whatever the character of the exudate, result in the production of vascular connective tissue; the existence of a bacony, indurated, semitransparent or opaque milky tissue in or about its an inflammatory area is is a distinct evidence of chronicity. What in- factors are responsible for this tissue formation cannot be definitely determined. The young cells derived from these fixed cells are capable of some degree of motility and indeed do move and the growing connective tissue cells and endothelial cells are forced to take the direction in which chemotactic and nutritive materials are located. The primary exudate, especially fibrin, possesses this power of attraction, the fibroblasts beneath a fibrinous covering being found actively proliferating and pushing into the fibrin. It is possible that the infiltration the cells, in other; of a tissue with exudate directly occasions an excessive nutrition of the cells, enabling them to form more protoplasm and then diloss of substance vide; and again the formation of spaces and the of the tissues should be considered, these factors favoring the de- velopment of inflammation, and having a tendency to increase physiological regeneration by release. The area of the proliferation depends upon the extent and duration of the inflam- mation. As already stated, the embryonic tissue appears in the form upon free surfaces (wounds, ulcers, fistulous passages, serous membranes). With a reddish-gray or fleshy red color, it presents a granular, uneven, undulating surface, from which in of granulations aseptic inflammation is given off a serous, reddish, viscid exudate, or when bacteria are present a purulent fluid. These intercapillary cells are leucocytes and fibroblasts, is usually spindle shaped. When there is motionless contact with an opposed and similarly inflamed surface, the two surfaces become adherent, at first by the embryonic tissue, but later by capillary anastomoses and the thorough interlacing of the developing tissue. Thus adhesive inflammation, the formation of adhesive bands, often in the form of broad connective tissue cords, results. The in- flammatory embryonic tissue gradually becomes pale and linn and 286 Inflammation. The bundles of fibrils continually growing thicker add firmness to the tissue, and with the cessation of inflam- mation the young tissue shrinks gradually to smaller bulk. Where, however, the irritation continues and final cicatrization is delayed the proliferaThis may tive changes sometimes take on an exuberant character. Indeed, interstitial proliferation of fibroblasts proceeding uninterruptedly for a long time may produce a mass of young inflammatory tissue far in excess of the original tissue it replaced and producing huge connective tissue enlargements (iibromatous inflammation, fibrous hypertrophy) On mucous membranes as that of the gall bladder, such proliferations meet but little resistance from the fluid in the cavity and often give rise to villous. Although the ordinary inflammatory irritants, the thermic, toxic and mechanical causes, act according to the intensity of their influence to cause that type of exudate, there are now this now some causative in- fluences which give rise to inflammatory reactions invariably having the same appearance and constant characteristics. Usually these types of disease are considered under special names, and will here be treated of in special chapters (v. The inflammatory processes almost exclusively run their course in the vascular connective the other constituents, gland cells, muscles or nerves, tissue of the organ; - take a more in or less passive part.

Location of centre of resistance and centre of rational using finite element method. A comparative cephlometric study of open bite cases & normal occlusion cases, Journal of Health Sciences Aug 2010 Page 364 2010 122. A simple clinical aid for placement of bonded retainer - Journal of Health Science Aug 2010 124. A comparative cephalometric study of open bite cases & normal occlusion cases - Journal of Health Science Dec 2010 125. Efficacy of lignocaine with clonidine and adrenaline in minor oral surgicprocedure Contemporary clinical dentistry Apr 2012 vol 3(2) 133. Efficacy of lignocaine with clonidine and adrenaline in minor oral surgical procedure Contemporary clinical dentistry Apr 2012 vol 3(2) 134. Continuing antiplatelet therapy throughout dental procedures -A clinical dilemma J Inter discip dent Mar 2012 136. Gorlin Goltz syndrome a case unearthed dental follicle e journal Jan 2012 2010-2011 137. Shyamala K- Risk of tumor cell seeding through biopsy and aspiration cytology-A technical analysis. Dietary habits and oral hygiene practices and prevalence of dental caries among 10-12 years old school children of south Bangalore. Perception of pictorial warning present on cigarette packet used in India among graduating dental students of Bangalore city. Management of hemi facial miscrostomia: Journal of Indian Prosthodontic Society, supplementary issue 2012, 138-141. A comparative evaluation of marginal adaptation of Zirconia copings and Ni-Cr coping using shoulder finish line design- an invitro study: Journal of Indian Prosthodontics Society. Oral rehabilitation and psychological management of hypohydrotic ectodermal dysplasia patients with partial anodontia. Anatomic basis for implant selection and positioning- a review: Journal of Oxford Dental College. A simplified approach for achieving harmonious occlusion in implant supported complete arch fixed prosthesis: International journal of oral implantology and clinical research. Fabricating bar for overdenture using wooden tooth picks with prefabricated metal clips and custom cast clips: Journal of Health Sciences and Research. Nanotechnology approaches to design better dental implant materials: Trends in biomaterials and artificial organs; vol 25, issue1; 2011 30-33. Obstructive sleep apnea and its prosthodontic management: Journal Of Health Sciences;2010,vol 1. Effect of pressure pot curing on properties of autopolymerizing acrylic resin: Journal Of Health Sciences;2010,vol, 5-8. Contemporary dental ceramics-an overview: Journal Of Health Sciences; vol 1:2010, 3845. Prosthodontic rehabilitation of patient with partial anodontia: A clinical report. Repeated fractures of implant supported mandibular over dentures opposing maxillary natural dentition.

Superficial temporal middle cerebral anastomosis (end-to-side) Giant middle cerebral aneurysm Trapping: Used for giant aneurysms (>25 mm diameter) where other methods have failed due to the width of the aneurysm neck. Should be combined with cerebral revascularisation to minimize the risk of ischaemia. A tracker catheter is inserted via a femoral puncture and guided up through the arterial system into the aneurysm sac. The coil attached to the end of a delivery wire is then guided into the fundus and after Posterior communicating aneurysm before and after coil embolisation accurate placement, the passage of an electric current causes electrochemical release. The radiologist aims to completely obliterate the fundus, but this is not always feasible and to avoid occluding the adjacent vessel, a portion of the neck may remain. In either case, a small risk of rebleeding persists, even when completely obliterated. A balloon is attached to a second catheter and periodically inflated across the aneurysm neck during coil insertion to preserve the vessel lumen. Coils are then packed into the fundus via a tracker catheter passed through the interstices of the stent. If tolerated, intra-arterial inflation of a detachable balloon can provide permanent occlusion. Intra-arterial balloon inflation can also provide temporary intra-operative protection when proximal control is difficult to achieve. Wide necked basilar aneurysm with one stent inserted into the left posterior cerebral artery, and another stent passing through the interstices of the first and inserted into the right posterior cerebral artery. Coil embolisation was reserved for aneurysms technically difficult to repair, particularly those in the posterior circulation. This swing occurred despite the trial being weighted towards small anterior circulation aneurysms in patients in good clinical condition. Long-term follow up (mean 9 years after treatment) of the trial patients has shown that although rebleeding was higher in the coil treatment group, the risk of death was still significantly lower in coiled patients. Aneurysm treatment requires a team approach involving interventional radiologists and neursurgeons. Treatment selection must take a variety of factors into account including the nature and location of the aneurysm, the relative difficulties of the endovascular or operative approach and the patients age and clinical condition. Unfortunately aneurysms that are difficult to treat with one technique are often difficult to treat with both methods. Calcium antagonists: several large studies and a meta-analysis have confirmed that Nimodipine reduces the incidence of cerebral infarction by about one third and improves outcome. High fluid intake (haemodilution): maintenance of a high fluid input (3 litres per day) may help prevent a fall in plasma volume from sodium and fluid loss. If hyponatraemia develops do not restrict fluids (this significantly increases the risk of cerebral infarction). If sodium levels fall below 130 mmol/1, give hypertonic saline or fludrocortisone. Plasma volume expansion (hypervolaemia): expanding the plasma volume with colloid. If clinical evidence of ischaemia develops despite this treatment, then (if the aneurysm has been repaired) combine with: Hypertensive therapy: treatment with inotropic agents. Since cerebral autoregulation commonly fails after subarachnoid haemorrhage, increasing blood pressure increases cerebral blood flow. Up to 70% of ischaemic neurological deficits developing after aneurysm operations can be reversed by inducing hypertension; often a critical level of blood pressure is evident. Early recognition and treatment of a developing neurological deficit may prevent progression from ischaemia to infarction.